CtrA controls cell division and outer membrane composition of the pathogen Brucella abortus
Brucella abortus Cell Cycle and Infection Are Coordinated
Trends Microbiol. (12):812-21.
G1-arrested newborn cells are the predominant infectious form of the pathogen Brucella abortus
Nature Communications 5, 4366
The Brucella pathogens are polarized bacteria
Microbes and Infection 15, 998-1004
BtaE, an adhesin that belongs to the trimeric autotransporter family, is required for full virulence and defines a specific adhesive pole of Brucella
Immun. 81, 996-1007
The histidine kinase PdhS controls cell cycle progression of the pathogenic alphaproteobacterium Brucella abortus
J. Bacteriol. 194, 5305-5314.
Characterization of a Brucella abortus mutant defective for the association with endoplasmic reticulum exit sites in HeLa cells
Microbiology 158, 2610-2618
Structural analysis of Brucella abortus RicA substitutions that do not impair interaction with human Rab2 GTPase
BMC Biochemistry 13, 16
Polar growth in the Alphaproteobacterial order Rhizobiales
Proc Natl. Acad. Sci. USA 109, 1697-1701
Identification of a Brucella spp. secreted protein specifically interacting with human Rab2 small GTPase
Cell. Microbiol. 13, 1044-1058
RpoE1, an extracytoplasmic function sigma factor, is a repressor of the flagellar system in Brucella melitensis
Microbiology 157, 1263-1268.
The alkylation response protein AidB is localized at the new poles and constriction sites in Brucella abortus
BMC Microbiol. 11, 257.
Functional characterization of the incomplete phosphotransferase system (PTS) of the intracellular pathogen Brucella melitensis
PLoS ONE 5, e12679.
Global analysis of quorum sensing targets in the intracellular pathogen Brucella melitensis 16M
J. Proteome Res. 9, 3200-3217.
Aggregates of soluble and folded proteins as intermediate state of inclusion body formation in Escherichia coli
BMC Microbiology 10, 248.
Identification of the essential Brucella melitensis porin Omp2b as a suppressor of Bax-induced cell death in yeast in a genome-wide screening
PLoS ONE 5, e13274.
PdhS, an old pole-localized histidine kinase, recruits the fumarase FumC in Brucella abortus
J. Bacteriol. 192, 3235-3239.
Intracellular rescuing of a B. melitensis 16M virB mutant by co-infection with a wild type strain
Microb. Pathog. 45, 134-141.
The asymmetric distribution of the essential histidine kinase PdhS indicates a differentiation event in Brucella abortus
EMBO J. 26, 1444-1455.
Mutations of the Quorum sensing-dependent regulator VjbR lead to drastic surface modifications in Brucella melitensis
J. Bacteriol. 189, 6035-6047.
FtcR is a new master regulator of the flagellar system of Brucella melitensis 16M with homologs in Rhizobiaceae
J. Bacteriol. 189, 131-141.
The stringent response mediator Rsh is required for Brucella spp. virulence, and regulates expression of the type IV secretion system VirB
Cell. Microbiol. 8, 1791-1802.
A RpoH-like heat shock sigma factor is involved in stress response and virulence in Brucella melitensis 16M
J. Bacteriol. 188, 7707-7710
NnrA is required for full virulence and regulates several Brucella melitensis denitrification genes
J. Bacteriol. 188, 1615-1619.
A quorum sensing regulator controls expression of both the type four secretion system and the flagellar apparatus of Brucella melitensis
Cell. Microbiol. 7, 1151-1161.
Systematic mutagenesis of Brucella melitensis 16M reveals a major role for GntR regulators in the control of virulence
Infect. Immun. 73, 5578-5586.
The Ton system, an ABC transporter, and a universally conserved GTPase are involved in iron utilization by Brucella melitensis 16M
Infect. Immun. 72, 5783-5790.
Morphological and functional asymmetry in alpha-proteobacteria
Trends Microbiol. 12, 361-365.
Generation of the Brucella melitensis ORFeome version 1.1
Genome Res. 14, 2201-2206.
Attenuated signature-tagged mutagenesis mutants of Brucella melitensis identified during the acute phase of infection in mice
Infect. Immun. 71, 7053-7060.
Prediction of proteins 3D structures
Bioinformatics 18, 1250-1256. IF: 3.4
Plasticity of transcriptional regulation network among alpha-proteobacteria is supported by the identification of CtrA targets in Brucella abortus
Mol. Microbiol. 43, 945-960. IF: 6.3